Matches in Nanopublications for { ?s ?p "[Based on these data, we conclude that accelerated in vitro apoptosis and increased Fas/ APO-1 and bcl-2 protein expression in SLE are nonspecific for the disease, and might be explained at least in part by the increased in vivo activation levels of PBMC from patients with SLE, MCTD, or autoimmune vasculitides combined with in vitro incubation under 'noninflammatory' conditions and growth factor withdrawal.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- assertion description "[Based on these data, we conclude that accelerated in vitro apoptosis and increased Fas/ APO-1 and bcl-2 protein expression in SLE are nonspecific for the disease, and might be explained at least in part by the increased in vivo activation levels of PBMC from patients with SLE, MCTD, or autoimmune vasculitides combined with in vitro incubation under 'noninflammatory' conditions and growth factor withdrawal.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Based on these data, we conclude that accelerated in vitro apoptosis and increased Fas/ APO-1 and bcl-2 protein expression in SLE are nonspecific for the disease, and might be explained at least in part by the increased in vivo activation levels of PBMC from patients with SLE, MCTD, or autoimmune vasculitides combined with in vitro incubation under 'noninflammatory' conditions and growth factor withdrawal.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP262769.RAB49yDYAVaMNH4nmiQNK1BijzY0b87xzBZCGOM5lEVEk130_assertion description "[Based on these data, we conclude that accelerated in vitro apoptosis and increased Fas/ APO-1 and bcl-2 protein expression in SLE are nonspecific for the disease, and might be explained at least in part by the increased in vivo activation levels of PBMC from patients with SLE, MCTD, or autoimmune vasculitides combined with in vitro incubation under 'noninflammatory' conditions and growth factor withdrawal.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP262769.RAB49yDYAVaMNH4nmiQNK1BijzY0b87xzBZCGOM5lEVEk130_provenance.
- NP248501.RA0_c9onQjczYdmJWODHBp4jp14GsQMucxHpFy0IwWqA0130_assertion description "[Based on these data, we conclude that accelerated in vitro apoptosis and increased Fas/ APO-1 and bcl-2 protein expression in SLE are nonspecific for the disease, and might be explained at least in part by the increased in vivo activation levels of PBMC from patients with SLE, MCTD, or autoimmune vasculitides combined with in vitro incubation under 'noninflammatory' conditions and growth factor withdrawal.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP248501.RA0_c9onQjczYdmJWODHBp4jp14GsQMucxHpFy0IwWqA0130_provenance.
- NP262381.RAVjwQcw0aWJKARHShDJdQvCV1aTFWWbhmuB8sTIFXqbs130_assertion description "[Based on these data, we conclude that accelerated in vitro apoptosis and increased Fas/ APO-1 and bcl-2 protein expression in SLE are nonspecific for the disease, and might be explained at least in part by the increased in vivo activation levels of PBMC from patients with SLE, MCTD, or autoimmune vasculitides combined with in vitro incubation under 'noninflammatory' conditions and growth factor withdrawal.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP262381.RAVjwQcw0aWJKARHShDJdQvCV1aTFWWbhmuB8sTIFXqbs130_provenance.