Matches in Nanopublications for { ?s ?p "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP294730.RAWBD0cG7eRe5CR7DmwXLNc3Mf19TKQ9kJM44fpmZtr-A130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP294730.RAWBD0cG7eRe5CR7DmwXLNc3Mf19TKQ9kJM44fpmZtr-A130_provenance.
- NP405800.RAgHYmdV32YtTP0B9vsUItL9WLTZVt4h_gcoIledLKjYs130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP405800.RAgHYmdV32YtTP0B9vsUItL9WLTZVt4h_gcoIledLKjYs130_provenance.
- NP182022.RADHezaXkH7RwKU7tvCI0iLpzK4y9tmRjV7udCvYFZ3jo130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP182022.RADHezaXkH7RwKU7tvCI0iLpzK4y9tmRjV7udCvYFZ3jo130_provenance.
- NP774746.RAoL-fk8HpTM95JAnTs_ZE3WLEeVYsQ39xkPU8alvQQHQ130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP774746.RAoL-fk8HpTM95JAnTs_ZE3WLEeVYsQ39xkPU8alvQQHQ130_provenance.
- NP137549.RATHjlLC110YqiqRBUmcM_3UtllAZIB-3WcMd9qsiy-Z4130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP137549.RATHjlLC110YqiqRBUmcM_3UtllAZIB-3WcMd9qsiy-Z4130_provenance.
- NP429208.RA4HLD-ZVDpTTI2L8tzP4gZQr9j5CVDqxlmhzOLee_mLA130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP429208.RA4HLD-ZVDpTTI2L8tzP4gZQr9j5CVDqxlmhzOLee_mLA130_provenance.
- NP782766.RA6o3mtPRwTSoruG4mdC-gyyUdmpj3T7YgUSTsjbeln6Y130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP782766.RA6o3mtPRwTSoruG4mdC-gyyUdmpj3T7YgUSTsjbeln6Y130_provenance.
- NP137757.RAyb3R7NJ6Viz2fhsPA5b4XU0bB9UuecwBoDYvLDZ6wYw130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP137757.RAyb3R7NJ6Viz2fhsPA5b4XU0bB9UuecwBoDYvLDZ6wYw130_provenance.
- NP69962.RAB_7Cgav9UCIo1x0BUUPlbKj_hcCWGsgF25GqDcj0Mj4130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP69962.RAB_7Cgav9UCIo1x0BUUPlbKj_hcCWGsgF25GqDcj0Mj4130_provenance.
- NP316495.RACfmKBom0evWcpiZPVxDiYpJW78mfW66agfr8OW5f1rA130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP316495.RACfmKBom0evWcpiZPVxDiYpJW78mfW66agfr8OW5f1rA130_provenance.
- NP440152.RAST7OfYE_Vg1rwO816LaVgRk6AFYgEkHFnoqPTzcvQUg130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP440152.RAST7OfYE_Vg1rwO816LaVgRk6AFYgEkHFnoqPTzcvQUg130_provenance.
- NP440663.RA3DgrgaSMJAPXE5gcaKnUUCXqxI7p1xk_En42jB-xTYs130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP440663.RA3DgrgaSMJAPXE5gcaKnUUCXqxI7p1xk_En42jB-xTYs130_provenance.
- assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP888230.RA1lYT_cVpUbceKb9sK5CGqfXLYIDzYYiQzM3oysu9bbM130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP888230.RA1lYT_cVpUbceKb9sK5CGqfXLYIDzYYiQzM3oysu9bbM130_provenance.
- NP888692.RA5kGEBUji1uvJMUSMOZSUFz5chMnu1YLejtIyM1TzJE0130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP888692.RA5kGEBUji1uvJMUSMOZSUFz5chMnu1YLejtIyM1TzJE0130_provenance.
- NP316494.RA4f4piaH4YbY1t6zmFf5uU68jgPJXuzdxWgHSDwFqXxs130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP316494.RA4f4piaH4YbY1t6zmFf5uU68jgPJXuzdxWgHSDwFqXxs130_provenance.
- NP69959.RAc2l4oxVj0juY13n9BdTGUMgdVhDpWTXA1IcJcNRnH5w130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP69959.RAc2l4oxVj0juY13n9BdTGUMgdVhDpWTXA1IcJcNRnH5w130_provenance.
- NP316500.RAROI5wfKaURJHdZVget8RNk_CzbcasxNF2GRE4WMNO4g130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP316500.RAROI5wfKaURJHdZVget8RNk_CzbcasxNF2GRE4WMNO4g130_provenance.
- NP316501.RAeXyY-TiFqCBv3zXWibZmiF4YVTIAyAOIZwOXcSyvZzM130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP316501.RAeXyY-TiFqCBv3zXWibZmiF4YVTIAyAOIZwOXcSyvZzM130_provenance.
- assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP316496.RAMbz59GT7XlTVDCcfz6x5Ksv258Fg0lPGa5MCsPU1Q0c130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP316496.RAMbz59GT7XlTVDCcfz6x5Ksv258Fg0lPGa5MCsPU1Q0c130_provenance.
- NP316499.RAJKOw2JOxUH8f9QnwShIz5E1wOh6xXPdo1pZ1u0n47Xg130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP316499.RAJKOw2JOxUH8f9QnwShIz5E1wOh6xXPdo1pZ1u0n47Xg130_provenance.
- NP437445.RAuP511bVsiAKZJfe_uz8M3vucoYzfaHs0tUcvB3lRtIM130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP437445.RAuP511bVsiAKZJfe_uz8M3vucoYzfaHs0tUcvB3lRtIM130_provenance.
- NP438106.RAuAWeBmwMZQWPPmGXaPb8pqZ_jDda3C8ENrxBVhkrx_8130_assertion description "[In this investigation, we explored the hypothesis that genetic polymorphisms in the cytochrome P4501A1 (T3801C) and glutathione S-transferase classes mu and theta (GSTM1 and GSTT1) gene deletions promote the development of cervical dysplasia by moderating the activation and detoxification of polycyclic hydrocarbons and other compounds that influence oxidative stress and DNA adduct formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP438106.RAuAWeBmwMZQWPPmGXaPb8pqZ_jDda3C8ENrxBVhkrx_8130_provenance.