Matches in Nanopublications for { ?s ?p "[It has been confirmed that increased production of reactive oxygen species (ROS) under various pathological conditions impairs HSC self-renewal and causes HSC premature exhaustion and BM suppression primarily via induction of HSC senescence, and oncogene induces accumulation of ROS and DNA damage and subsequently cellular senescence, which functions as an important barrier to prevent the growth of transformed cells to form a neoplasia.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP325394.RAQOwStSuujV63s3EHSt36P9kidqG1IARijoAkgtNWDFo130_assertion description "[It has been confirmed that increased production of reactive oxygen species (ROS) under various pathological conditions impairs HSC self-renewal and causes HSC premature exhaustion and BM suppression primarily via induction of HSC senescence, and oncogene induces accumulation of ROS and DNA damage and subsequently cellular senescence, which functions as an important barrier to prevent the growth of transformed cells to form a neoplasia.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP325394.RAQOwStSuujV63s3EHSt36P9kidqG1IARijoAkgtNWDFo130_provenance.
- NP414458.RA91NNT_Fu6TkFQ_ZY8qmubHpppQTIqw5q9YZEI397pxA130_assertion description "[It has been confirmed that increased production of reactive oxygen species (ROS) under various pathological conditions impairs HSC self-renewal and causes HSC premature exhaustion and BM suppression primarily via induction of HSC senescence, and oncogene induces accumulation of ROS and DNA damage and subsequently cellular senescence, which functions as an important barrier to prevent the growth of transformed cells to form a neoplasia.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP414458.RA91NNT_Fu6TkFQ_ZY8qmubHpppQTIqw5q9YZEI397pxA130_provenance.
- NP414559.RA_kwCZxK7DR3i4xEBw9xlOkWoSe4l3jKr1maipCpTA7U130_assertion description "[It has been confirmed that increased production of reactive oxygen species (ROS) under various pathological conditions impairs HSC self-renewal and causes HSC premature exhaustion and BM suppression primarily via induction of HSC senescence, and oncogene induces accumulation of ROS and DNA damage and subsequently cellular senescence, which functions as an important barrier to prevent the growth of transformed cells to form a neoplasia.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP414559.RA_kwCZxK7DR3i4xEBw9xlOkWoSe4l3jKr1maipCpTA7U130_provenance.