Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP1213271.RAD4PD1ETnMNsHzF_pEnrLumzPr_f5M_8T_mWvxmkFw0M130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1213271.RAD4PD1ETnMNsHzF_pEnrLumzPr_f5M_8T_mWvxmkFw0M130_provenance.
• NP744607.RAdiLdsGeSwrqryyhLVSIzNThsjr9EZkzg7X-tLg3eAmQ130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP744607.RAdiLdsGeSwrqryyhLVSIzNThsjr9EZkzg7X-tLg3eAmQ130_provenance.
• NP744621.RAUxKNMjoaxWytiBxZA30I1GvK4SnCzS4Uxyb7ihQogdk130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP744621.RAUxKNMjoaxWytiBxZA30I1GvK4SnCzS4Uxyb7ihQogdk130_provenance.
• NP744594.RA7Qfg6Bpi-EEVCJV5xYEpdbe51GXST3mywVuFtT7Cbyk130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP744594.RA7Qfg6Bpi-EEVCJV5xYEpdbe51GXST3mywVuFtT7Cbyk130_provenance.
• NP497618.RAIJCjjQHXyQtRoRx2uCS9Qgea7cAjWMdFNcvgvQlwB7Q130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP497618.RAIJCjjQHXyQtRoRx2uCS9Qgea7cAjWMdFNcvgvQlwB7Q130_provenance.
• NP746493.RAuTwdZ2ZwoON4cSlcBSau1a4L3HwHUPGwWgnImm32LyE130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP746493.RAuTwdZ2ZwoON4cSlcBSau1a4L3HwHUPGwWgnImm32LyE130_provenance.
• NP1213259.RAlU9z_LYVSrOKxPFCFe8UG0CFKq21AYcSpKnldQzMUAA130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1213259.RAlU9z_LYVSrOKxPFCFe8UG0CFKq21AYcSpKnldQzMUAA130_provenance.
• NP1213268.RAkLZqSuKGKX-I1gdb0Ykww3w2Lb3hGl709D79Yp_Jowo130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1213268.RAkLZqSuKGKX-I1gdb0Ykww3w2Lb3hGl709D79Yp_Jowo130_provenance.
• NP1213272.RAo7fRjGiTOOYrVq-S3RGZ3fIUxy0R312jQd5bbyLS93s130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1213272.RAo7fRjGiTOOYrVq-S3RGZ3fIUxy0R312jQd5bbyLS93s130_provenance.
• NP1213273.RAuY4j8Zey2BKvxBFgXGSSBLvgD2y1q-Yl-1M86cILg9c130_assertion description "[Our results suggested that aberrant activated autophagy in macrophages contributed to the pathogenesis of murine lupus possibly via promoting the production of proinflammatory cytokines TNF-α and IL-6, and inhibition of autophagy might represent a novel regulation strategy for excessive activation of proinflammatory macrophages and a new therapeutic regime for SLE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1213273.RAuY4j8Zey2BKvxBFgXGSSBLvgD2y1q-Yl-1M86cILg9c130_provenance.