Matches in Nanopublications for { ?s ?p "[Studies using gene-modified mice deficient in Nox2 activity indicate that Nox2 activation contributes to angiotensin II-induced cardiomyocyte hypertrophy, atrial fibrillation, and the development of interstitial fibrosis but may also positively modulate physiological excitation-contraction coupling.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- assertion description "[Studies using gene-modified mice deficient in Nox2 activity indicate that Nox2 activation contributes to angiotensin II-induced cardiomyocyte hypertrophy, atrial fibrillation, and the development of interstitial fibrosis but may also positively modulate physiological excitation-contraction coupling.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP540226.RAfsUqcfmJiOZGJl4c0BkaxXBfHGkeTamJ4TZBTcEI-Fc130_assertion description "[Studies using gene-modified mice deficient in Nox2 activity indicate that Nox2 activation contributes to angiotensin II-induced cardiomyocyte hypertrophy, atrial fibrillation, and the development of interstitial fibrosis but may also positively modulate physiological excitation-contraction coupling.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP540226.RAfsUqcfmJiOZGJl4c0BkaxXBfHGkeTamJ4TZBTcEI-Fc130_provenance.
- assertion description "[Studies using gene-modified mice deficient in Nox2 activity indicate that Nox2 activation contributes to angiotensin II-induced cardiomyocyte hypertrophy, atrial fibrillation, and the development of interstitial fibrosis but may also positively modulate physiological excitation-contraction coupling.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.