Matches in Nanopublications for { ?s ?p "[The modulating role of mannose binding lectin in the occurrence of cirrhosis in cystic fibrosis could be explained by the fact that hepatotoxic damage from viruses or bacteria might be increased by the immunodeficiency associated with mannose binding lectin variants and might facilitate the degradation of liver status.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP239018.RArL4qS4J1djQz7hHbfTSIhcc9zXJnti_1YLZkwUEd69s130_assertion description "[The modulating role of mannose binding lectin in the occurrence of cirrhosis in cystic fibrosis could be explained by the fact that hepatotoxic damage from viruses or bacteria might be increased by the immunodeficiency associated with mannose binding lectin variants and might facilitate the degradation of liver status.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP239018.RArL4qS4J1djQz7hHbfTSIhcc9zXJnti_1YLZkwUEd69s130_provenance.
- NP547239.RA22LVa-qglIqHx-Niq9z3NROjbjckukzdM5HTTPyTkbE130_assertion description "[The modulating role of mannose binding lectin in the occurrence of cirrhosis in cystic fibrosis could be explained by the fact that hepatotoxic damage from viruses or bacteria might be increased by the immunodeficiency associated with mannose binding lectin variants and might facilitate the degradation of liver status.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP547239.RA22LVa-qglIqHx-Niq9z3NROjbjckukzdM5HTTPyTkbE130_provenance.
- NP547727.RA9acjyR8cqOCIytJUiahIMgeTsUNFbOLr1r_81jTc3JY130_assertion description "[The modulating role of mannose binding lectin in the occurrence of cirrhosis in cystic fibrosis could be explained by the fact that hepatotoxic damage from viruses or bacteria might be increased by the immunodeficiency associated with mannose binding lectin variants and might facilitate the degradation of liver status.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP547727.RA9acjyR8cqOCIytJUiahIMgeTsUNFbOLr1r_81jTc3JY130_provenance.
- NP316708.RA3nQIdCIDGTcnwQSkmFW0LH06sFEvYTsn8vwbNeKAC88130_assertion description "[The modulating role of mannose binding lectin in the occurrence of cirrhosis in cystic fibrosis could be explained by the fact that hepatotoxic damage from viruses or bacteria might be increased by the immunodeficiency associated with mannose binding lectin variants and might facilitate the degradation of liver status.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP316708.RA3nQIdCIDGTcnwQSkmFW0LH06sFEvYTsn8vwbNeKAC88130_provenance.
- NP316709.RA1gMqNTBnCatv7rwElThXXD7ZVEcf2ByX4PrzbnsAMcU130_assertion description "[The modulating role of mannose binding lectin in the occurrence of cirrhosis in cystic fibrosis could be explained by the fact that hepatotoxic damage from viruses or bacteria might be increased by the immunodeficiency associated with mannose binding lectin variants and might facilitate the degradation of liver status.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP316709.RA1gMqNTBnCatv7rwElThXXD7ZVEcf2ByX4PrzbnsAMcU130_provenance.
- assertion description "[The modulating role of mannose binding lectin in the occurrence of cirrhosis in cystic fibrosis could be explained by the fact that hepatotoxic damage from viruses or bacteria might be increased by the immunodeficiency associated with mannose binding lectin variants and might facilitate the degradation of liver status.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The modulating role of mannose binding lectin in the occurrence of cirrhosis in cystic fibrosis could be explained by the fact that hepatotoxic damage from viruses or bacteria might be increased by the immunodeficiency associated with mannose binding lectin variants and might facilitate the degradation of liver status.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.