Matches in Nanopublications for { ?s ?p "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP874092.RAXMfTPMNenUUe06p4LEzsuYjY7BQK54NC5UDB99rS_CQ130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP874092.RAXMfTPMNenUUe06p4LEzsuYjY7BQK54NC5UDB99rS_CQ130_provenance.
- NP845592.RAk8aEB9nQWAqzQlkSSk7C8F5x4VVUulbVqRSITmm_h8w130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP845592.RAk8aEB9nQWAqzQlkSSk7C8F5x4VVUulbVqRSITmm_h8w130_provenance.
- NP334010.RA4Bgu69Fae52GfDKQ2isp8-_PIYP02nfHDl6ng2ssEIU130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP334010.RA4Bgu69Fae52GfDKQ2isp8-_PIYP02nfHDl6ng2ssEIU130_provenance.
- NP556513.RAFPz_FV8WFk-N8JxJeWpNhIlejGgOLmQUrEWaqo8gKbo130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP556513.RAFPz_FV8WFk-N8JxJeWpNhIlejGgOLmQUrEWaqo8gKbo130_provenance.
- assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP607448.RAx-ybn37bk9bPxRDqS6QyzooPLJroKlXPkMn5DSIHMAQ130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP607448.RAx-ybn37bk9bPxRDqS6QyzooPLJroKlXPkMn5DSIHMAQ130_provenance.
- NP376505.RAIAjb4u-XdZirxo2OFdBVT61HePSOODitsr1HQnm0ruw130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP376505.RAIAjb4u-XdZirxo2OFdBVT61HePSOODitsr1HQnm0ruw130_provenance.
- assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP607443.RAJ-xmISmeYnLlnmfZacPhVMWm8JyzD0xV--bFQw-tejg130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP607443.RAJ-xmISmeYnLlnmfZacPhVMWm8JyzD0xV--bFQw-tejg130_provenance.
- NP732056.RAo-99tr210ZpB75DBuKgRr4hF8CxOuqZfw2NtE4C82sI130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP732056.RAo-99tr210ZpB75DBuKgRr4hF8CxOuqZfw2NtE4C82sI130_provenance.
- NP607442.RAsVNZMh-Vjaq5MLh0cekpteNcpMZGipdMMD9mE9QZVlA130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP607442.RAsVNZMh-Vjaq5MLh0cekpteNcpMZGipdMMD9mE9QZVlA130_provenance.
- NP607446.RAnW8gYdDlAIpNBK9Pe_nUWqSH9mM179iEf4i1RYacFjs130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP607446.RAnW8gYdDlAIpNBK9Pe_nUWqSH9mM179iEf4i1RYacFjs130_provenance.
- NP607449.RAj0VlCMj7o-EVhHHPoydm8_C4Y6ZGSttuGSqvQYDXvLo130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP607449.RAj0VlCMj7o-EVhHHPoydm8_C4Y6ZGSttuGSqvQYDXvLo130_provenance.
- NP607451.RAmCQC_TWKrigRl5_f2QQuauOvYTBEcVB2NS1eHz5Rx3s130_assertion description "[The neoplastic EC cell is characterized by loss of TGFbeta-1-mediated growth inhibition and, similar to glioblastomas, utilizes the TGFbeta system to induce gene responses associated with growth promotion (c-Myc and the ERK pathway), invasion (E-cadherin), and metastasis (MTA1).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP607451.RAmCQC_TWKrigRl5_f2QQuauOvYTBEcVB2NS1eHz5Rx3s130_provenance.