Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP410987.RADdRmy9NJrPn4QdWxN3rI_vjYR2yJ2ObogP8hRm7bv7w130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP410987.RADdRmy9NJrPn4QdWxN3rI_vjYR2yJ2ObogP8hRm7bv7w130_provenance.
• NP419495.RAAf_r4cmIqvQ71ANzTyf5WqMekx-48Tp5bcCuwDBhK3I130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419495.RAAf_r4cmIqvQ71ANzTyf5WqMekx-48Tp5bcCuwDBhK3I130_provenance.
• NP449140.RAYGFprOERct4hbrgyw2ob6cwhMUFS40FFjtel_NG3YGU130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP449140.RAYGFprOERct4hbrgyw2ob6cwhMUFS40FFjtel_NG3YGU130_provenance.
• NP449851.RAaQacgXxOodgTXspnu93oxg9BPc36rJmn6pPssFHVJCc130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP449851.RAaQacgXxOodgTXspnu93oxg9BPc36rJmn6pPssFHVJCc130_provenance.
• NP481234.RA2fYJJ5m4Yu5xPtnwT3xKXUkD9isvhwgSCsMyBGs6gpc130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP481234.RA2fYJJ5m4Yu5xPtnwT3xKXUkD9isvhwgSCsMyBGs6gpc130_provenance.
• NP520011.RA408tafIgKVAA1jblOBDpdJOEnPZsqkUfheqhp-gBAxs130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP520011.RA408tafIgKVAA1jblOBDpdJOEnPZsqkUfheqhp-gBAxs130_provenance.
• NP520457.RA1DIHp-iYBCjBrPrss0SnBo5ar7D2WF4yHZ8GdBPilZw130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP520457.RA1DIHp-iYBCjBrPrss0SnBo5ar7D2WF4yHZ8GdBPilZw130_provenance.
• NP520698.RA1yEh43Px5lHOl5KmJAJ4arUMnx3VV2TnKJHVchvmqFw130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP520698.RA1yEh43Px5lHOl5KmJAJ4arUMnx3VV2TnKJHVchvmqFw130_provenance.
• NP520343.RAdI4RCkYyP9swhttQRrTo9ZEazeXuePWtCFaLRdKDotg130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP520343.RAdI4RCkYyP9swhttQRrTo9ZEazeXuePWtCFaLRdKDotg130_provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP419489.RA3JRVsxFrxJu_spH3LYxZSVhNynUFv4ZiGQXEpUGrib0130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419489.RA3JRVsxFrxJu_spH3LYxZSVhNynUFv4ZiGQXEpUGrib0130_provenance.
• NP419496.RA_t3mCi_Rlqk1B2PRA0eLBgI1bQcESXsNOYqBisMEgtU130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419496.RA_t3mCi_Rlqk1B2PRA0eLBgI1bQcESXsNOYqBisMEgtU130_provenance.
• NP419499.RA9Nq7LGvYh4LrP9Ylg91NAe1liy4zM2aRNJ4s7Fbc-rg130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419499.RA9Nq7LGvYh4LrP9Ylg91NAe1liy4zM2aRNJ4s7Fbc-rg130_provenance.
• NP419491.RARBf-MANZJiwweLAzH-JldjN2fXP2tWgHa-uNAL7WhLw130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419491.RARBf-MANZJiwweLAzH-JldjN2fXP2tWgHa-uNAL7WhLw130_provenance.
• NP419493.RAeCzbkCiNR0rsXzofM7kmxZNsfakph3Q0hiQmkLmrlkU130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419493.RAeCzbkCiNR0rsXzofM7kmxZNsfakph3Q0hiQmkLmrlkU130_provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP410806.RAEkcCdpytfyONTxRJljkfEZfjGOCl2408f-MAUfZEnBQ130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP410806.RAEkcCdpytfyONTxRJljkfEZfjGOCl2408f-MAUfZEnBQ130_provenance.
• NP520592.RAHBoAX0cy970_KxlWXmB_CfS5la79dIOC4nXZny49zCQ130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP520592.RAHBoAX0cy970_KxlWXmB_CfS5la79dIOC4nXZny49zCQ130_provenance.
• assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP419490.RAMfZZsgQniC6sbJ8dkjoAC3cy_jDRgw_-QDioAI1achU130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419490.RAMfZZsgQniC6sbJ8dkjoAC3cy_jDRgw_-QDioAI1achU130_provenance.
• NP419492.RAOaJt89Upw2JZBZowu6ZntpCescbPqCwT6lGdPyHxk7w130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419492.RAOaJt89Upw2JZBZowu6ZntpCescbPqCwT6lGdPyHxk7w130_provenance.
• NP410608.RAkF4H_1wqIX5SDePzuQdGpyHVaJsY4DBz_rkDx7ldCPg130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP410608.RAkF4H_1wqIX5SDePzuQdGpyHVaJsY4DBz_rkDx7ldCPg130_provenance.
• NP411100.RAgxyZ_IiS6BXu6judaJkCX1Kl185L8Es9adlVkdziy3Q130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP411100.RAgxyZ_IiS6BXu6judaJkCX1Kl185L8Es9adlVkdziy3Q130_provenance.
• NP520181.RAkWjd-CyEHZkvz1W8ceMp3KQPt_O8doVi23FYIJla3Ts130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP520181.RAkWjd-CyEHZkvz1W8ceMp3KQPt_O8doVi23FYIJla3Ts130_provenance.
• NP419485.RAsV3WIZMXF6m5HRzlsEL9fhyTGSxjUzNnnA66bhKZuFk130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419485.RAsV3WIZMXF6m5HRzlsEL9fhyTGSxjUzNnnA66bhKZuFk130_provenance.
• NP419488.RAvPL-Bmti3yPC1YfzFjNaKcq4D3pP7JM04JEx5oaqeeY130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419488.RAvPL-Bmti3yPC1YfzFjNaKcq4D3pP7JM04JEx5oaqeeY130_provenance.
• NP419494.RApkAlffPcVVG9zq5sxO1V91ny71duZ9bEG_RrujkyCdg130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419494.RApkAlffPcVVG9zq5sxO1V91ny71duZ9bEG_RrujkyCdg130_provenance.
• NP419497.RAujCazuXpKwOhpsNVD49b3limWufWnj41hlyDm1HtJ3M130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419497.RAujCazuXpKwOhpsNVD49b3limWufWnj41hlyDm1HtJ3M130_provenance.
• NP419498.RApFV9SqMjUXq3Z7lFXBI1qeiN6jeOj5TSaNkQ6fVWuJw130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419498.RApFV9SqMjUXq3Z7lFXBI1qeiN6jeOj5TSaNkQ6fVWuJw130_provenance.
• NP419500.RAtABUPFLvm3bcU1LezX_AJfTng2yfRRQ7CITohQVfTZ0130_assertion description "[These data indicate that IGF-I stimulates VEGF synthesis in thyroid carcinomas in an Akt-dependent pathway via AP-1 and HIF-1 alpha and provide the framework for clinical use of small-molecule inhibitors, including geldanamycin analogs, to abrogate proangiogenic cascades in thyroid cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP419500.RAtABUPFLvm3bcU1LezX_AJfTng2yfRRQ7CITohQVfTZ0130_provenance.