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Matches in Nanopublications for { ?s ?p "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP750912.RAXNKmOjwZBfqGWCm8nKng2oDf2PHl_veK2Su3VI3crOo130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP750912.RAXNKmOjwZBfqGWCm8nKng2oDf2PHl_veK2Su3VI3crOo130_provenance.
• NP756170.RAVbP8u-S5EjvyZl0jwYtji4EpUaj8nu4qbCdi_24EjYg130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP756170.RAVbP8u-S5EjvyZl0jwYtji4EpUaj8nu4qbCdi_24EjYg130_provenance.
• NP236965.RACSg2w022JTH-ZTqH7jvz2NpIsicLrz1re7zZ9W7KNuE130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP236965.RACSg2w022JTH-ZTqH7jvz2NpIsicLrz1re7zZ9W7KNuE130_provenance.
• NP687250.RANjyNnORxT9iB___bkUawzDingyiE71vZkRmJiH0ZJ08130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP687250.RANjyNnORxT9iB___bkUawzDingyiE71vZkRmJiH0ZJ08130_provenance.
• NP384520.RABjIUgCuVAkmYRtmH1o6Rnkr7wvPhu4lsVrOdDC86oPg130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP384520.RABjIUgCuVAkmYRtmH1o6Rnkr7wvPhu4lsVrOdDC86oPg130_provenance.
• NP441884.RAeqx0Tm8Av5bz-mehQvMC1llQFsyB2mYUa2gXhvwV-WI130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP441884.RAeqx0Tm8Av5bz-mehQvMC1llQFsyB2mYUa2gXhvwV-WI130_provenance.
• assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP384521.RA4DtVfXoCnvAaFLSrDDtruNwiT2KO0MeBN34G24C6TGQ130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP384521.RA4DtVfXoCnvAaFLSrDDtruNwiT2KO0MeBN34G24C6TGQ130_provenance.
• NP384522.RAXppUqm5C3hiAI9zeSMPAxW21VrC70RGQ8BxL0hnagCI130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP384522.RAXppUqm5C3hiAI9zeSMPAxW21VrC70RGQ8BxL0hnagCI130_provenance.
• NP441932.RAi55AgIKerOGCX-zLhvhCZMBB172oEuC2bPXXLXGFowo130_assertion description "[This chapter focuses on recent data obtained from biochemical and electrophysiological studies of GCAP1/GCAP2 knockout mice and other GCAP transgenic mice, addressing: 1. the quantitative aspects of the Ca2+-feedback to Ret-GCs in regulating the light sensitivity and adaptation in intact rods; 2. functional differences between GCAP1 and GCAP2 in intact rod photoreceptors; and 3. whether GCAP mutants with impaired Ca2+ binding lead to retinal disease in vivo by constitutive activation of Ret-GCs and elevation of intracellular cGMP, as predicted from in vitro studies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP441932.RAi55AgIKerOGCX-zLhvhCZMBB172oEuC2bPXXLXGFowo130_provenance.