Matches in Nanopublications for { ?s ?p "[Treatment of human hepatocytes with resistin at levels observed in human obesity stimulated apoB secretion up to 10-fold, because of increased microsomal triglyceride transfer protein (MTP) activity and decreased expression/phosphorylation of proteins in the insulin signaling pathways (insulin receptor substrate-2, Akt, and extracellular signal-regulated kinase).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP282164.RAUwAFiTdrbFpc9KhQylKGLK-use5JTX3nF8nC1Ujs8n8130_assertion description "[Treatment of human hepatocytes with resistin at levels observed in human obesity stimulated apoB secretion up to 10-fold, because of increased microsomal triglyceride transfer protein (MTP) activity and decreased expression/phosphorylation of proteins in the insulin signaling pathways (insulin receptor substrate-2, Akt, and extracellular signal-regulated kinase).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP282164.RAUwAFiTdrbFpc9KhQylKGLK-use5JTX3nF8nC1Ujs8n8130_provenance.
- NP476076.RAlxmNKJ_mmZWKNlOC998mFknbFglmqCiK3a8XuIaqSxk130_assertion description "[Treatment of human hepatocytes with resistin at levels observed in human obesity stimulated apoB secretion up to 10-fold, because of increased microsomal triglyceride transfer protein (MTP) activity and decreased expression/phosphorylation of proteins in the insulin signaling pathways (insulin receptor substrate-2, Akt, and extracellular signal-regulated kinase).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP476076.RAlxmNKJ_mmZWKNlOC998mFknbFglmqCiK3a8XuIaqSxk130_provenance.
- NP828197.RAlFUUbVqSruhawBcjGcuz_1NgMjG4Lizz44fNohyPLkU130_assertion description "[Treatment of human hepatocytes with resistin at levels observed in human obesity stimulated apoB secretion up to 10-fold, because of increased microsomal triglyceride transfer protein (MTP) activity and decreased expression/phosphorylation of proteins in the insulin signaling pathways (insulin receptor substrate-2, Akt, and extracellular signal-regulated kinase).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP828197.RAlFUUbVqSruhawBcjGcuz_1NgMjG4Lizz44fNohyPLkU130_provenance.
- assertion description "[Treatment of human hepatocytes with resistin at levels observed in human obesity stimulated apoB secretion up to 10-fold, because of increased microsomal triglyceride transfer protein (MTP) activity and decreased expression/phosphorylation of proteins in the insulin signaling pathways (insulin receptor substrate-2, Akt, and extracellular signal-regulated kinase).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Treatment of human hepatocytes with resistin at levels observed in human obesity stimulated apoB secretion up to 10-fold, because of increased microsomal triglyceride transfer protein (MTP) activity and decreased expression/phosphorylation of proteins in the insulin signaling pathways (insulin receptor substrate-2, Akt, and extracellular signal-regulated kinase).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.