Matches in Nanopublications for { ?s ?p "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP625630.RAZuAf-HV2DN8_j9udb1QRXP0IDhGKNSnK3L6YOf8aRsE130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP625630.RAZuAf-HV2DN8_j9udb1QRXP0IDhGKNSnK3L6YOf8aRsE130_provenance.
- NP623641.RA0Z0KrFdiZCIapYFOBu7wouq0SMhI2LJAe_k1n3Ml68Y130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP623641.RA0Z0KrFdiZCIapYFOBu7wouq0SMhI2LJAe_k1n3Ml68Y130_provenance.
- NP1158966.RA8U6Mpev8vIxbvJEj1-B68WxyfhMXPtqCcqCPrzIxL_Q130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1158966.RA8U6Mpev8vIxbvJEj1-B68WxyfhMXPtqCcqCPrzIxL_Q130_provenance.
- NP1158972.RA2IoyO59wBpoeoCbU-CdETa1cXNDMeoqiRg-1LD1ubLs130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1158972.RA2IoyO59wBpoeoCbU-CdETa1cXNDMeoqiRg-1LD1ubLs130_provenance.
- NP1158973.RA8EQjtiPE6uZtfoYphe0o8Qm96UGXvLG-aZjOjpx6BRg130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1158973.RA8EQjtiPE6uZtfoYphe0o8Qm96UGXvLG-aZjOjpx6BRg130_provenance.
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- assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP622326.RAuD5lk0PYzGt6K13rdFoqxkNbpmLg60nQRIN3Qb3BWwQ130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP622326.RAuD5lk0PYzGt6K13rdFoqxkNbpmLg60nQRIN3Qb3BWwQ130_provenance.
- NP623173.RAp_LWXt7CxHf-gEo5rFSUHl7Cku9GZoeDda3TxQ8Vayc130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP623173.RAp_LWXt7CxHf-gEo5rFSUHl7Cku9GZoeDda3TxQ8Vayc130_provenance.
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- NP624772.RAiGbHwDJrH5Cv_X1mxdmXHrZejUgimM1f28yu_YzwyAk130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP624772.RAiGbHwDJrH5Cv_X1mxdmXHrZejUgimM1f28yu_YzwyAk130_provenance.
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- NP1158971.RAuyAChqop-NZSiRjCqUmZ08pwKo5qxpkrsNEodh4jefc130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1158971.RAuyAChqop-NZSiRjCqUmZ08pwKo5qxpkrsNEodh4jefc130_provenance.
- NP1158976.RAjgi4jFqllRW5RM92qSQVuQRrT7KMic9GXKIRUbvkdiY130_assertion description "[We characterized the mechanisms by which a novel Akt kinase inhibitor MK-2206 induced cell death in IGF1R-dependent colorectal cancer (CRC) cells with upregulated PI3K/Akt signaling in response to IGF1R activation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1158976.RAjgi4jFqllRW5RM92qSQVuQRrT7KMic9GXKIRUbvkdiY130_provenance.