Matches in Nanopublications for { ?s ?p "[We have analyzed the somatic mutations in 89 cutaneous neurofibromas derived from three unrelated NF1 patients with high tumor burden, by loss of heterozygosity (LOH) analysis of the NF1, TP53, RB1, and CDKN2A genes, by assessing microsatellite instability (MSI), by direct sequencing of the NF1, TP53, and several mismatch repair (MMR) genes and by multiplex ligation-dependent probe amplification of the NF1 and TP53 genes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP392093.RAD8Ky9udxHiEOKlWF6Z9CspjY5XekGW99Au8t6qB4jSk130_assertion description "[We have analyzed the somatic mutations in 89 cutaneous neurofibromas derived from three unrelated NF1 patients with high tumor burden, by loss of heterozygosity (LOH) analysis of the NF1, TP53, RB1, and CDKN2A genes, by assessing microsatellite instability (MSI), by direct sequencing of the NF1, TP53, and several mismatch repair (MMR) genes and by multiplex ligation-dependent probe amplification of the NF1 and TP53 genes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP392093.RAD8Ky9udxHiEOKlWF6Z9CspjY5XekGW99Au8t6qB4jSk130_provenance.
- NP237111.RArusIx3hAsGqS85dF8vHLI4k0erjapjO7OpQ2SNzYXuo130_assertion description "[We have analyzed the somatic mutations in 89 cutaneous neurofibromas derived from three unrelated NF1 patients with high tumor burden, by loss of heterozygosity (LOH) analysis of the NF1, TP53, RB1, and CDKN2A genes, by assessing microsatellite instability (MSI), by direct sequencing of the NF1, TP53, and several mismatch repair (MMR) genes and by multiplex ligation-dependent probe amplification of the NF1 and TP53 genes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP237111.RArusIx3hAsGqS85dF8vHLI4k0erjapjO7OpQ2SNzYXuo130_provenance.
- NP743678.RAQEWt9Yp_L7yD3p5Rfbv52Glj0D4SgmSLSgIj-tgcQV8130_assertion description "[We have analyzed the somatic mutations in 89 cutaneous neurofibromas derived from three unrelated NF1 patients with high tumor burden, by loss of heterozygosity (LOH) analysis of the NF1, TP53, RB1, and CDKN2A genes, by assessing microsatellite instability (MSI), by direct sequencing of the NF1, TP53, and several mismatch repair (MMR) genes and by multiplex ligation-dependent probe amplification of the NF1 and TP53 genes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP743678.RAQEWt9Yp_L7yD3p5Rfbv52Glj0D4SgmSLSgIj-tgcQV8130_provenance.
- NP462654.RAVNdspYG30OZNgG_ocYXx82okCB8i-5IQrVOXrv8_oa4130_assertion description "[We have analyzed the somatic mutations in 89 cutaneous neurofibromas derived from three unrelated NF1 patients with high tumor burden, by loss of heterozygosity (LOH) analysis of the NF1, TP53, RB1, and CDKN2A genes, by assessing microsatellite instability (MSI), by direct sequencing of the NF1, TP53, and several mismatch repair (MMR) genes and by multiplex ligation-dependent probe amplification of the NF1 and TP53 genes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP462654.RAVNdspYG30OZNgG_ocYXx82okCB8i-5IQrVOXrv8_oa4130_provenance.
- assertion description "[We have analyzed the somatic mutations in 89 cutaneous neurofibromas derived from three unrelated NF1 patients with high tumor burden, by loss of heterozygosity (LOH) analysis of the NF1, TP53, RB1, and CDKN2A genes, by assessing microsatellite instability (MSI), by direct sequencing of the NF1, TP53, and several mismatch repair (MMR) genes and by multiplex ligation-dependent probe amplification of the NF1 and TP53 genes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We have analyzed the somatic mutations in 89 cutaneous neurofibromas derived from three unrelated NF1 patients with high tumor burden, by loss of heterozygosity (LOH) analysis of the NF1, TP53, RB1, and CDKN2A genes, by assessing microsatellite instability (MSI), by direct sequencing of the NF1, TP53, and several mismatch repair (MMR) genes and by multiplex ligation-dependent probe amplification of the NF1 and TP53 genes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.