Matches in Nanopublications for { ?s ?p "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP206791.RAZMpVqNtes41PjGkP9sUyBjZRpKI6z1onpnuHAPsxqCU130_assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP206791.RAZMpVqNtes41PjGkP9sUyBjZRpKI6z1onpnuHAPsxqCU130_provenance.
- NP397453.RAiBJUeQQdHwZNCFOmqJ-YpjlQdS7eAVYsMUO2jPTfVvU130_assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP397453.RAiBJUeQQdHwZNCFOmqJ-YpjlQdS7eAVYsMUO2jPTfVvU130_provenance.
- assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP398179.RAGVzf69xuwu3PLYkKg8d7GVLaMm8sr2FaZ_LCzV_PaMs130_assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP398179.RAGVzf69xuwu3PLYkKg8d7GVLaMm8sr2FaZ_LCzV_PaMs130_provenance.
- NP260071.RA3s_gT6IuieACmUnnSPMknsRE4Otd6goZN0XcnBJ3P3M130_assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP260071.RA3s_gT6IuieACmUnnSPMknsRE4Otd6goZN0XcnBJ3P3M130_provenance.
- assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP739167.RA8TbeJGKSQniToHOu5uPhSsa5LAIVEtHRmg4MSatoSZw130_assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP739167.RA8TbeJGKSQniToHOu5uPhSsa5LAIVEtHRmg4MSatoSZw130_provenance.
- NP611372.RAMPi10ugzu7SQEWwCP5xaf3B9G3MnSHkwHEcxbVcNZPs130_assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP611372.RAMPi10ugzu7SQEWwCP5xaf3B9G3MnSHkwHEcxbVcNZPs130_provenance.
- NP739159.RAUbCzDIKzkitb3ZZClt7H8H3Avl1sJQxU0xIXnEa1t7Q130_assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP739159.RAUbCzDIKzkitb3ZZClt7H8H3Avl1sJQxU0xIXnEa1t7Q130_provenance.
- NP739168.RAZkV63q6TPAKwf043AzNJzW_imjFuahgBYqo2DZuvqUg130_assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP739168.RAZkV63q6TPAKwf043AzNJzW_imjFuahgBYqo2DZuvqUg130_provenance.
- assertion description "[We show that mono-allelic loss of pten in the probasin-driven-ErbB-2 model resulted in increased nuclear cyclin D1 and proliferating cell nuclear antigen levels and decreased disease latency compared to either individual genetic model and, unlike the probasin-driven-ErbB-2 mice, progression to adenocarcinoma.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.