Matches in Nanopublications for { ?s ?p "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" ?g. }
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- _4 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _6 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _6 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _4 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _3 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _6 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _4 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _4 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _6 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _6 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _4 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _5 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.
- _5 value "Treatment with palmitate rapidly induced the association of myeloid differentiation factor 88 (MyD88) with the TLR2 receptor, activated the stress-linked kinases p38, JNK, and protein kinase C, induced degradation of IkappaBalpha, and increased NF-kappaB DNA binding...TLR2 mediates the initial events of fatty acid-induced insulin resistance in muscle" provenance.