Matches in Nanopublications for { ?s ?p ?o <http://rdf.disgenet.org/resource/nanopub/NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance>. }
Showing items 1 to 9 of
9
with 100 items per page.
- source_evidence_literature type ECO_0000212 NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.
- befree-2016 importedOn "2016-02-19" NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.
- NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_assertion wasGeneratedBy ECO_0000203 NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.
- NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_assertion wasDerivedFrom befree-2016 NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.
- NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_assertion SIO_000772 23075332 NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.
- NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_assertion evidence source_evidence_literature NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.
- NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_assertion description "[The present review discusses the substrates of FXIIIa (activated FXIII) involved in thrombosis and wound healing with a particular focus on: (i) the influence of plasma FXIIIa on the formation of stable fibrin clots able to withstand mechanical and enzymatic breakdown through fibrin-fibrin cross-linking and cross-linking of fibrinolysis inhibitors, in particular α2-antiplasmin; (ii) the role of intracellular FXIIIa in clot retraction through cross-linking of platelet cytoskeleton proteins, including actin, myosin, filamin and vinculin; (iii) the role of intracellular FXIIIa in cross-linking the cytoplasmic tails of monocyte AT1Rs (angiotensin type 1 receptors) and potential effects on the development of atherosclerosis; and (iv) the role of FXIIIa on matrix deposition and tissue repair, including cross-linking of extracellular matrix proteins, such as fibronectin, collagen and von Willebrand factor, and the effects on matrix deposition and cell-matrix interactions.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1021838.RASN9JVWgfQ77v0oJEiMW9c5euEQmKEddhjUQ_xDyvZJ4130_provenance.