Matches in Nanopublications for { ?s ?p ?o <http://rdf.disgenet.org/resource/nanopub/NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance>. }
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- source_evidence_literature type ECO_0000212 NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.
- befree-2016 importedOn "2016-02-19" NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.
- NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_assertion wasGeneratedBy ECO_0000203 NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.
- NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_assertion wasDerivedFrom befree-2016 NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.
- NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_assertion SIO_000772 19082521 NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.
- NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_assertion evidence source_evidence_literature NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.
- NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_assertion description "[Diabetes-associated variants in CDKAL1, CDKN2B, HHEX/IDE, IGF2BP2, KCNJ11, SLC30A8 and TCF7L2 are associated with physiological alterations leading to T2DM, such as glucose intolerance, impaired insulin secretion or insulin resistance, supporting their role in the disease aetiology.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP709320.RAlT9Bnw8VwHMYOY_TpjYRdmzE4QYevmU2Ejq_e1hc9x4130_provenance.