Matches in Nanopublications for { ?s ?p ?o <http://rdf.disgenet.org/resource/nanopub/NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance>. }
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- source_evidence_literature type ECO_0000212 NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.
- befree-20150227 importedOn "2015-02-27" NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.
- NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_assertion wasGeneratedBy ECO_0000203 NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.
- NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_assertion wasDerivedFrom befree-20150227 NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.
- NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_assertion SIO_000772 21891868 NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.
- NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_assertion evidence source_evidence_literature NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.
- NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_assertion description "[In fact, Nectin-2 (NC-2); apolipoprotein E (APOE); glycoprotein carcinoembryonic antigen related cell adhesion molecule-16 (CEACAM-16); B-cell lymphoma-3 (Bcl-3); translocase of outer mitochondrial membrane 40 homolog (T0MM-40); complement receptor-1 (CR-l); APOJ or clusterin and C-type lectin domain A family-16 member (CLEC-16A); Phosphatidyl inositol- binding clathrin assembly protein gene (PICALM); ATP-bonding cassette, sub family A, member 7 (ABCA7); membrane spanning A4 (MSA4); CD2 associated protein (CD2AP); cluster of differentiation 33 (CD33); and ephrin receptor A1 (EPHA1) result in a genetic signature that might affect individual brain susceptibility to infection by the herpes virus family during aging, leading to neuronal loss, inflammation, and amyloid deposition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP841213.RAAHHy1HyNzX7vDc-FsU_QMl5e4zAlYE4Nuq41yPv6HH8130_provenance.