Matches in Nanopublications for { ?s ?p ?o <http://www.tkuhn.ch/bel2nanopub/RA--TnF0FEmCGPo5CQDsA6eDcCx5_BXoPU70pu1MQUXJY#provenance>. }
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- _7 label "Selventa" provenance.
- large_corpus.bel title "BEL Framework Large Corpus Document" provenance.
- large_corpus.bel description "Approximately 61,000 statements." provenance.
- large_corpus.bel version "1.4" provenance.
- large_corpus.bel authoredBy _7 provenance.
- assertion wasDerivedFrom large_corpus.bel provenance.
- assertion wasDerivedFrom _6 provenance.
- assertion hadPrimarySource 17341548 provenance.
- _6 wasQuotedFrom 17341548 provenance.
- _6 value "Glucose increased c-Myc abundance and association with its heterodimer partner Max. Manipulations that prevented the formation of a functional c-Myc/Max heterodimer reduced the expression of the L-PK gene. In addition, glucose augmented the binding of ChREBP, c-Myc, and Max to the promoter of the L-PK gene in situ. #[JL] Chrebp is MLXIPL, and is implicated as a glucose-responsive factor. This is basically saying that glucose activates MLXIPL- activation of MYC" provenance.
- large_corpus.bel rights "Copyright (c) 2011-2012, Selventa. All rights reserved." provenance.