Matches in Nanopublications for { ?s ?p ?o <http://www.tkuhn.ch/bel2nanopub/RADjTXS34u2POvCCk2I_uB5UMWKC9uJXpdqD68RCZv2Ek#provenance>. }
Showing items 1 to 11 of
11
with 100 items per page.
- _5 label "Selventa" provenance.
- large_corpus.bel title "BEL Framework Large Corpus Document" provenance.
- large_corpus.bel description "Approximately 61,000 statements." provenance.
- large_corpus.bel version "20131211" provenance.
- large_corpus.bel authoredBy _5 provenance.
- assertion wasDerivedFrom large_corpus.bel provenance.
- assertion wasDerivedFrom _4 provenance.
- assertion hadPrimarySource 11956243 provenance.
- _4 wasQuotedFrom 11956243 provenance.
- _4 value "# Pubmed:gene: Increased vascular permeability in C1 inhibitor-deficient mice mediated by the bradykinin type 2 receptor. This increased vascular permeability was reversed by treatment with intravenous human C1INH, with a Kunitz domain plasma kallikrein inhibitor (DX88), and with a bradykinin type 2 receptor (Bk2R) antagonist (Hoe140). In addition, treatment of the C1INH-deficient mice with an angiotensin-converting enzyme inhibitor (captopril) increased the vascular permeability. Bradykinin is a small peptide vasodilator formed in blood and tissue fluids by cleavage of alpha2-globulins. It causes both arteriolar dilation and increased capillary permeability" provenance.
- large_corpus.bel rights "Copyright (c) 2011-2012, Selventa. All rights reserved." provenance.