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- _4 label "Selventa" provenance.
- large_corpus.bel title "BEL Framework Large Corpus Document" provenance.
- large_corpus.bel description "Approximately 61,000 statements." provenance.
- large_corpus.bel version "20131211" provenance.
- large_corpus.bel authoredBy _4 provenance.
- assertion wasDerivedFrom large_corpus.bel provenance.
- assertion wasDerivedFrom _3 provenance.
- assertion hadPrimarySource 12626582 provenance.
- _3 wasQuotedFrom 12626582 provenance.
- _3 value "In this study, we report that S100A8, S100A9, and S100A8/A9 caused neutrophil chemotaxis at concentrations of 10(-12)-10(-9) M. S100A8, S100A9, and S100A8/A9 stimulated shedding of L-selectin, up-regulated and activated Mac-1, and induced neutrophil adhesion to fibrinogen in vitro. As shown in Fig. 3, MRPs induced shedding of L-selectin (CD62L) from the neutrophil surface. In contrast, expression of CD11b, but not CD51 was up-regulated following stimulation with MRPs. As Mac-1 is stored in specific and gelatinase granules, this suggests that MRPs induced neutrophil degranulation." provenance.
- large_corpus.bel rights "Copyright (c) 2011-2012, Selventa. All rights reserved." provenance.