Matches in Nanopublications for { ?s ?p "[We conclude that polymorphisms within the NOD Ica1 core promoter may determine AIRE-mediated down-regulation of ICA69 expression in medullary thymic epithelial cells, thus providing a novel mechanistic explanation for the loss of immunologic tolerance to this self-antigen in autoimmunity.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP215832.RAtJYfwjpx8uPsFwEejn3W_hGnpKbzFvzBn2lO0Gr6L2U130_assertion description "[We conclude that polymorphisms within the NOD Ica1 core promoter may determine AIRE-mediated down-regulation of ICA69 expression in medullary thymic epithelial cells, thus providing a novel mechanistic explanation for the loss of immunologic tolerance to this self-antigen in autoimmunity.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP215832.RAtJYfwjpx8uPsFwEejn3W_hGnpKbzFvzBn2lO0Gr6L2U130_provenance.
- NP429026.RAEngcLgNIwf5HX1bbKh4YMVceAjfQJGv8mssRhaqEH3k130_assertion description "[We conclude that polymorphisms within the NOD Ica1 core promoter may determine AIRE-mediated down-regulation of ICA69 expression in medullary thymic epithelial cells, thus providing a novel mechanistic explanation for the loss of immunologic tolerance to this self-antigen in autoimmunity.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP429026.RAEngcLgNIwf5HX1bbKh4YMVceAjfQJGv8mssRhaqEH3k130_provenance.
- assertion description "[We conclude that polymorphisms within the NOD Ica1 core promoter may determine AIRE-mediated down-regulation of ICA69 expression in medullary thymic epithelial cells, thus providing a novel mechanistic explanation for the loss of immunologic tolerance to this self-antigen in autoimmunity.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We conclude that polymorphisms within the NOD Ica1 core promoter may determine AIRE-mediated down-regulation of ICA69 expression in medullary thymic epithelial cells, thus providing a novel mechanistic explanation for the loss of immunologic tolerance to this self-antigen in autoimmunity.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.