Matches in Nanopublications for { ?s ?p "Glucose increased c-Myc abundance and association with its heterodimer partner Max. Manipulations that prevented the formation of a functional c-Myc/Max heterodimer reduced the expression of the L-PK gene. In addition, glucose augmented the binding of ChREBP, c-Myc, and Max to the promoter of the L-PK gene in situ. #[JL] Chrebp is MLXIPL, and is implicated as a glucose-responsive factor. This is basically saying that glucose activates MLXIPL- activation of MYC" ?g. }
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- _6 value "Glucose increased c-Myc abundance and association with its heterodimer partner Max. Manipulations that prevented the formation of a functional c-Myc/Max heterodimer reduced the expression of the L-PK gene. In addition, glucose augmented the binding of ChREBP, c-Myc, and Max to the promoter of the L-PK gene in situ. #[JL] Chrebp is MLXIPL, and is implicated as a glucose-responsive factor. This is basically saying that glucose activates MLXIPL- activation of MYC" provenance.
- _6 value "Glucose increased c-Myc abundance and association with its heterodimer partner Max. Manipulations that prevented the formation of a functional c-Myc/Max heterodimer reduced the expression of the L-PK gene. In addition, glucose augmented the binding of ChREBP, c-Myc, and Max to the promoter of the L-PK gene in situ. #[JL] Chrebp is MLXIPL, and is implicated as a glucose-responsive factor. This is basically saying that glucose activates MLXIPL- activation of MYC" provenance.