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- NP138345.RAo0_65d9gQ48-TvYD2aJWJs8nYcQFLo4VWGsNUYHwU8g130_assertion type Assertion NP138345.RAo0_65d9gQ48-TvYD2aJWJs8nYcQFLo4VWGsNUYHwU8g130_head.
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- NP138345.RAo0_65d9gQ48-TvYD2aJWJs8nYcQFLo4VWGsNUYHwU8g130_assertion SIO_000772 21041608 NP138345.RAo0_65d9gQ48-TvYD2aJWJs8nYcQFLo4VWGsNUYHwU8g130_provenance.
- NP138345.RAo0_65d9gQ48-TvYD2aJWJs8nYcQFLo4VWGsNUYHwU8g130_assertion evidence source_evidence_literature NP138345.RAo0_65d9gQ48-TvYD2aJWJs8nYcQFLo4VWGsNUYHwU8g130_provenance.
- NP138345.RAo0_65d9gQ48-TvYD2aJWJs8nYcQFLo4VWGsNUYHwU8g130_assertion description "[Genetic variation in AKT1 may mediate both short-term as well as longer-term effects on psychosis expression associated with use of cannabis, possibly through a mechanism of cannabinoid-regulated AKT1/GSK-3 signaling downstream of the dopamine D(2) recept]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP138345.RAo0_65d9gQ48-TvYD2aJWJs8nYcQFLo4VWGsNUYHwU8g130_provenance.