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- NP629736.RAvJLhIDqu8hg1An6l8E7bsc5pyS5J_q3-rmRjsL2BU9U130_assertion type Assertion NP629736.RAvJLhIDqu8hg1An6l8E7bsc5pyS5J_q3-rmRjsL2BU9U130_head.
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- NP629736.RAvJLhIDqu8hg1An6l8E7bsc5pyS5J_q3-rmRjsL2BU9U130_assertion SIO_000772 23689131 NP629736.RAvJLhIDqu8hg1An6l8E7bsc5pyS5J_q3-rmRjsL2BU9U130_provenance.
- NP629736.RAvJLhIDqu8hg1An6l8E7bsc5pyS5J_q3-rmRjsL2BU9U130_assertion evidence source_evidence_literature NP629736.RAvJLhIDqu8hg1An6l8E7bsc5pyS5J_q3-rmRjsL2BU9U130_provenance.
- NP629736.RAvJLhIDqu8hg1An6l8E7bsc5pyS5J_q3-rmRjsL2BU9U130_assertion description "[Taken together, these results suggest that the abnormal upregulation of PLD1 may contribute to the pathogenesis of IL-1β-induced chronic arthritis and that a selective PLD1 inhibitor might provide a potential therapeutic molecule for the treatment of chronic inflammatory autoimmune disorders.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP629736.RAvJLhIDqu8hg1An6l8E7bsc5pyS5J_q3-rmRjsL2BU9U130_provenance.