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{ <http://www.tkuhn.ch/bel2nanopub/RA-RniytLtf3jXCscpGWXjPrBT_EofIPMVH-RgYB1GR2M#_5> ?p ?o ?g. }
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"
CpG ODN also increased NFkappaB activity, and a potent inhibitor of NFkappaB that significantly blocked CpG-induced Nox1 expression, suggesting that Nox1 regulation is mediated through an NFkappaB-dependent mechanism.
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