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{ <http://www.tkuhn.ch/bel2nanopub/RA0ufE5EbS3_D9wG_YDEoSTfQ-LzC27ciuDk5APFAHDdM#_2> ?p ?o ?g. }
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MtDNA damage, in turn, leads to increased ROS production and atherogenesis. 20 For example, mutations in complex I genes lead to increased mitochondrial ROS production.21,22
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