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{ <http://www.tkuhn.ch/bel2nanopub/RA5FRpLHEufIgI8_WTri8L9LZCRLbcY0lxkp_Gn4eW6fA#_4> ?p ?o ?g. }
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"
Hyperoxia-dependent ROS production was strongly reduced in lung sections, in isolated epithelial type II cells, and lung endothelial cells from NOX1-deficient mice.
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