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{ <http://www.tkuhn.ch/bel2nanopub/RA64kfExqA2h_A33YjtIGBKTeUOL6V8z66Qkg5Hi9N7N4#_3> ?p ?o ?g. }
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"
ROS levels were significantly elevated in hypoxic HepG2 cells, and anti-oxidants prevented the hypoxic down-regulation of hepcidin. Conversely, a prooxidant, H(2)O(2), suppressed hepcidin expression in these cells even in normoxia.
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