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{ <http://www.tkuhn.ch/bel2nanopub/RA9gLIt_q2hHt0PjhQKKZiP2QrENxPDomO1txuumSbhew#_6> ?p ?o ?g. }
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"
We speculate that loss of AKR1C1 and AKR1C2 in breast cancer results in decreased progesterone catabolism, which, in combination with increased PR expression, may augment progesterone signaling by its nuclear receptors.
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