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{ <http://www.tkuhn.ch/bel2nanopub/RABcFdk7eXiM_nnx-lC_JznYZ0qwhDjPkmJOOEh-0LJ9M#_3> ?p ?o ?g. }
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In fact, induction of a UPR in the setting of oxidant stress promotes cell survival, whereas knockdown or knockout of GRP78, calreticulin, or PDI increases ROS burden and promotes cell death (21, 52, 53).
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