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{ <http://www.tkuhn.ch/bel2nanopub/RAJM5aiQRN6SwfjuQndvKDi-_Yo4F1ZZ1XyDHmYS0cCQ4#_7> ?p ?o ?g. }
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As shown in Fig. 4E, ectopic expression of wild-type AKT2 significantly enhanced UV- and TNFalpha-induced NFkB activity, which was abolished by treatment of cells with LY294002 or dominant negative AKT2.
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