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{ <http://www.tkuhn.ch/bel2nanopub/RAKilTcJpmRI9LVWyFp1JoKyVixnL3zb_X8o8bTdhFhJw#_4> ?p ?o ?g. }
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cIAP ablation prevents RIP1 ubiquitination and favors the formation of an alternative complex II, containing RIP1, FADD, and caspase- 8, which leads to RIP1 kinase–dependent activation of caspase-8 and subsequent apoptosis (37, 46).
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