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{ <http://www.tkuhn.ch/bel2nanopub/RAMjF_DXEC8PMsVw3v8xIyNe8ZgymLeGm4KtXI99koHl0#_5> ?p ?o ?g. }
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These effects appear to be protein kinase A-dependent. Furthermore inhibition of protein kinase C restored the beta(3)-receptor-mediated reductions in insulin-induced IRS-1 tyrosine phosphorylation and IRS-1-associated PI3-kinase activity
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