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{ <http://www.tkuhn.ch/bel2nanopub/RAO_B3kr6LRRLdDRtk2vq04k_jbdbu40H-MY8k6a5Z838#_7> ?p ?o ?g. }
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"
Expression of wild-type PDK1 significantly inhibited H2O2-induced capase-3 activity and PARP cleavage in a dosedependent manner, whereas there was no effect in the presence of kinase-dead PDK1 (supplemental Fig. 4).
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