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{ <http://www.tkuhn.ch/bel2nanopub/RAQH9fCF38OpyY657D30itaLDf9Z-w7Q1AsErHL-Zr-jU#_3> ?p ?o ?g. }
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"
Furthermore, angiotensin II increased the proportion of senescent VSMCs and induced the expression of proinflammatory molecules, as well as p21, in aortic atherosclerotic lesions in a mouse model of atherosclerosis.33
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