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{ <http://www.tkuhn.ch/bel2nanopub/RAWXTNWjw2zurrsLq0AoAlQAKWOp1Mj-uPcBggrh_hWMI#_4> ?p ?o ?g. }
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Both the growth-stimulating activity of low-dose NO for endothelial cells and suppression of TSP1 expression were ERK-dependent. Conversely, exogenous TSP1 suppressed NO-mediated pERK
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