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{ <http://www.tkuhn.ch/bel2nanopub/RAZmD5NWDgL-srjrBU9ds08oTfsIGM9Lbxf9GYVMU7dBw#_5> ?p ?o ?g. }
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Stable transfection of wild type PS1 but not PS2 corrected EGFR to levels comparable to PS +/+ cells, while FAD PS1 mutations showed partial loss of activity. The C-terminal fragment of PS1 was sufficient to fully reduce EGFR levels.
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