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{ <http://www.tkuhn.ch/bel2nanopub/RAcEQLi4ak1IrOhLO4Z1vLEZTiGFehrpbHy_Z261bmPUc#_4> ?p ?o ?g. }
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"
Sin1 over-expression enhanced osmotic stress-induced phosphorylation of ATF-2 and ATF-2-mediated transcription, whereas knockdown of Sin1 expression by siRNA suppressed these responses.
"
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