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{ <http://www.tkuhn.ch/bel2nanopub/RAcGYk8L40_yNTZXFOpo27h3-3w3AyrKXuFoWh9fuSyjA#_4> ?p ?o ?g. }
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"
AKT inactivation induces mitochondrial impairment associated with opening of the permeability transition pore but is not dependent on the activation state of bad, glycogen synthase kinase-3beta, mammalian target of rapamycin, or bcl-xL
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