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{ <http://www.tkuhn.ch/bel2nanopub/RAhK-MPtl3TNpaJFE_cs4MbGuR1BQr3Qtd4STy73CPJec#_5> ?p ?o ?g. }
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"
The enhanced FA utilization induced by FoxO1 was mediated by a severalfold increase in plasma membrane level of the fatty acid translocase FAT/CD36 and eliminated by cell treatment with the CD36 inhibitor sulfo-N-succinimidyl-oleate.
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