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{ <http://www.tkuhn.ch/bel2nanopub/RAju7BdX-i1xIiHOegonA-aCFCSqeetl24BfNMop-PMPs#_3> ?p ?o ?g. }
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The expression of NOX-3 was increased because of loss of Toll-like receptor 4. Furthermore, these mice had increased alveolar cell apoptosis and loss of antielastolytic properties in lung lysates
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