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{ <http://www.tkuhn.ch/bel2nanopub/RAkv6p_a8er76nO1Hs4WM7TqH6TqCQn2qEnZDFx3Oe3_Y#_4> ?p ?o ?g. }
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In MEFs, stimulation of the IGF1 receptor exerts antiapoptotic actions via activation of PI-3 kinase and Akt by a mechanism that is independent of the intrinsic tyrosine kinase activity of the receptor (Povsic et al., 2003).
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