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{ <http://www.tkuhn.ch/bel2nanopub/RAnFpZODFjUgtXT4a09HxvehS-GmS8jt1QnWp588ksPoE#_5> ?p ?o ?g. }
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The treatment of NCI-N87 cells with EGF resulted in increases in phosphorylation of Erk1/2, Akt, and P38. Blockade of the Erk, phosphatidylinositol-3 kinase/Akt, or P38 pathways in this cell line
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