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- _2 wasQuotedFrom 16870827 provenance.
- _2 value "Recent data implicates ERK (p42/44 kinase) in ANG II-mediated VSMC contraction. Touyz et al. (192) showed that in VSMCs from human peripheral arteries, tyrosine kinases and the ERK signaling cascade play a role in Ca2+ and pHi pathways, which ultimately cause cell contraction; specifically, MEK/ERK may increase Ca2+ availability within cells. " provenance.