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{ <http://www.tkuhn.ch/bel2nanopub/RApP7rreN4FLw8UDxR1EUGOiw3eTp8enPBKOquBwnszHE#_3> ?p ?o ?g. }
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EPO-enhanced Pl3K activity was suppressed by WT treatment in a dose-dependent manner and constant inhibition of Pl3K by WT interfered with both hemoglobin synthesis and glycophorin A expression promoted by EPO
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