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{ <http://www.tkuhn.ch/bel2nanopub/RApU6u_E5FbejhUpqcdrzCjcETlANwS_lO944MeThelR8#_5> ?p ?o ?g. }
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Mechanistically, ALK1-dependent SMAD signaling synergizes with activated Notch in stalk cells to induce expression of the Notch targets HEY1 and HEY2, thereby repressing VEGF signaling, tip cell formation, and endothelial sprouting
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