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{ <http://www.tkuhn.ch/bel2nanopub/RAyaE2hnd0q4D1fFVOq5MKaS_Al40MUw-vhfuf41Gv1mQ#_3> ?p ?o ?g. }
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We confirmed the ER stress-induced activation of NF-k B and AP-1 using EMSA. Each of the ER stress reagents (2-deoxyglucose, 2-DG; inhibitor of glycosylation) showed the activation of NF-k B and AP-1 (Fig. 3A).
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